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Schminke U, Hilker L, Motsch L, Griewing B, Kessler C: Volumetric assessment of plaque progression with 3-dimensional ultrasonography under statin therapy. J Neuroimaging 12: 245–251 (2002). Shaalan WE, Cheng H, Gewertz B, McKinsey JF, Schwartz LB, Katz D, Cao D, Desai T, Glagov S, Bassiouny HS: Degree of carotid plaque calcification in relation to symptomatic outcome and plaque inflammation. J Vasc Surg 40: 262–269 (2004). : The diagnostic accuracy of ex vivo MRI for human atherosclerotic plaque characterization.

45] Mackay J, Mensah G: Atlas of Heart Disease and Stroke. World Health Organization (2004). [46] Manca G, Parenti G, Bellina R, Boni G, Grosso M, Bernini W, Palombo C, Paterni M, Pelosi G, Lanza M, Mazzuca N, Bianchi R, De Caterina R: 111 In platelet scintigraphy for the noninvasive detection of carotid plaque thrombosis. Stroke 32: 719–727 (2001). : Angiogenesis and the atherosclerotic carotid plaque: an association between symptomatology and plaque morphology. J Vasc Surg 30: 261–268 (1999).

60–69%). Recent studies demonstrate that Doppler criteria are influenced by equipment used [11], laboratories [2] and the technologist performing the test [24]. Additionally, contralateral disease has been associated 37 with increased carotid volume flow resulting in an overestimation of the severity of disease [31]. Most diagnostic laboratories simply use the well-established criteria of others to diagnose severe carotid stenosis. However, for the reasons cited above, it is recommended that each laboratory validate its own Doppler criteria for clinically relevant stenoses [18].

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